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Athlete’s foot, or tinea pedis, is a skin disease caused by a fungal infection.  The infection typically occurs between the toes, and the feet are most subject to this disease because shoes best create the warm, dark, and moist environment in which fungus thrives.  Other areas that create a similar environment, such as swimming pools, public showers, and locker rooms; can also promote fungi growth. 

Symptoms of athlete’s foot include dry skin, itching, scaling, inflammation, and blistering.  Sometimes, blisters can evolve into the cracks or breaks in the skin.  The exposed tissue can then create pain, swelling, and discharge.  The spread of infection can cause itching and burning as well.

While athlete’s foot commonly occurs between the toes, it may also spread to the toenails or soles of the feet.  Other parts of the body, such as the groin or underarms, can also become infected if they are touched after the original area of infection is scratched.  Aside from physical contact, athlete’s foot can also spread through the contamination of footwear, clothing or bedsheets.

Proper foot hygiene is essential in preventing athlete’s foot.  You can prevent the fungus from spreading by frequently washing your feet using soap and water, thoroughly drying the feet between the toes, changing shoes and socks every day to reduce moisture, and ensuring that bathroom and shower floors are disinfected.  Other tips include using shower shoes, avoiding walking barefoot in public environments, wearing light and airy shoes, and wearing socks that keep the feet dry.

While treatment for athlete’s foot can involve topical or oral antifungal drugs, mild cases of the infection can be treated by dusting foot powder in shoes and socks.  Any treatment used can be supplemented by frequently bathing the feet and drying the toes.  If proper foot hygiene and self-care do not ease your case of athlete’s foot, contact your podiatrist.  He will determine if the underlying cause of your condition is truly a fungus.  If that is the case, a comprehensive treatment plan may be suggested with the inclusion of prescription antifungal medications.

Heel spurs are calcium deposits that cause bone protrusions on the heel bone. Heel spurs are usually associated with plantar fasciitis, which occurs when the plantar fasciitis in the foot becomes inflamed. Typically, heel spurs don’t cause any symptoms. However, they can produce chronic or intermittent heel pain. Those who have had the condition often describe the irritation as a stabbing pain.

There are risk factors that may make you more likely to develop heel spurs. People who have abnormal walking gaits, run and jog on hard surfaces, are obese, or wear poorly fitting shoes are more likely to develop heel spurs.

Fortunately, there are precautions you can take to avoid developing heel spurs. One of the best ways to do this is by wearing well-fitting shoes with shock-absorbent soles. Another preventative technique is to choose running shoes if you plan on running, and walking shoes if you plan on walking. Shoes are made for different activities and it is important to research a shoe before you purchase a pair.

The pain associated with heel spurs often decreases the more you walk. However, a recurrence of pain after an extended period of rest or walking is likely to occur with this condition. Those with severe heel spur pain may opt to go the surgical route for treatment.  However, more than 90% of those with the condition get better without surgical treatment. If you have a heel spur and want to know if surgery is right for you, you should go to your podiatrist and he or she will be able to conduct a pre-surgical test or exam to determine if you are an optimal candidate for surgery.

Gout, typically found in diabetic patients, is an unusually painful form of arthritis caused by elevated levels of uric acid in the bloodstream. The condition typically strikes the big joint on the big toe. It has also been known to strike the knees, elbows, fingers, ankles and wrists—generally anywhere that has a functioning, moving joint.

The high level of uric acid in a person’s bloodstream creates the condition known as hyperuricema—the main cause of gout. Genetic predisposition occurs in nine out of ten sufferers. The children of parents who suffer gout will have a two in ten chance of developing the condition as well. 

This form of arthritis, being particularly painful, is the leftover uric acid crystallizing in the blood stream. The crystallized uric acid then travels to the space between joints where they rub, causing friction when the patient moves. Symptoms include: pain, redness, swelling, and inflammation. Additional side effects may include fatigue and fever, although reports of these effects are very rare. Some patients have reported that pain may intensify when the temperature drops, such as when you sleep.

Most cases of gout are easily diagnosed by a podiatrist’s assessment of the various symptoms. Defined tests can also be performed. A blood test to detect elevated levels of uric acid is often used as well as an x-ray to diagnose visible and chronic gout.

Treatment for gout simply means eliminating symptoms. Non-steroid anti-inflammatory drugs or NSAIDs (Colchicine and other corticosteroid drugs, etc.) will quell the redness, the swelling, and the inflammation. However, managing your diet, lifestyle changes, and using preventative drugs are all helpful toward fully combating the most severe cases.

 Those that lead an inactive lifestyle are at a higher risk for gout. Any amount of exercise decreases the probability of repeat encounters with the condition. Reducing your consumption of red meat, sea food, and fructose-sweetened drinks also reduces the likelihood of chronic gout as well.

Ingesting Vitamin C, coffee, and particular dairy products can help with maintaining a healthy lifestyle. There are new drugs out on the market that inhibit the body’s production of uric acid-producing enzymes. However, reducing or eliminating your overall levels of uric acid is the best remedy to ensuring you lead a gout-free life.

The ankle is a hinged synovial joint made up of three bones: the tibia (shin bone), the fibula (outer ankle bone), and the talus (between the heel and leg). These three bones are bound, supported, and stabilized by strong, fibrous bands of tissue called ligaments.

A break in an ankle bone can be either traumatic or stress related. This injury may be referred to as a break or fracture. A traumatic fracture can result from tripping, twisting or rolling the ankle, falling, or by blunt impact to the ankle. These traumatic ankle breaks usually occur during sporting activities or accidents. Stress fractures, however, occur over time and are the result of repetitive stress to the ankle. These fractures sometimes occur when a new activity that engages the ankle is introduced, or when the level of activity is abruptly increased or intensified. 

There are various symptoms that accompany an ankle break. The most significant symptoms are pain and swelling that occurs in the ankle and sometimes spreads up from the foot to below the knee. Bruising or discoloration may develop eventually. It will be difficult or even impossible to put weight on the affected foot, and in severe cases there may be a visible deformity or even exposed bone.

It is very important to seek immediate treatment when an ankle break occurs or is suspected to have occurred, in order to allow the bone to properly heal and to avoid future complications such as stiff joints, limited range of motion, and osteoarthritis.

To diagnose a broken ankle, your podiatrist will first ask you to explain how the injury occurred and what your symptoms are. They will perform a thorough examination, checking for damage to nerves, blood vessels, and other structures around the injury site. They will also test your range of motion. An X-ray will need to be reviewed and, in some cases, an MRI or CT scan may be necessary.

Proper treatment of a broken ankle will depend on where and how severe the break is, how stable the ankle is, and whether the bone is displaced (misaligned or separated) or non-displaced (broken yet still aligned properly). 

Mild fractures (where the bone is non-displaced) may be treated by resting, icing, and elevating the ankle at first, followed by immobilization with a cast or walking boot. Pain and inflammation may be treated with acetaminophen. More severe or complicated fractures where bones or joints are displaced may require surgery. 

Recovery time will also vary, and it may take 4-6 weeks or longer for a broken ankle to heal. Your podiatrist will most likely order progressive X-rays or stress tests to be taken in order to monitor the healing process.